Friday, October 24, 2014


English: Street in Kenema, Sierra Leone.
English: Street in Kenema, Sierra Leone. (Photo credit: Wikipedia)

Ebola Liberia

Why has the Ebola virus suddenly erupted in a region of Africa known as the “Meningitis Belt” (comprising all four countries affected – Guinea, Liberia, Nigeria & Sierra Leone)?

Because the WHO & CDC are deliberately trying to cover up their bloody tracks.

The recent Ebola outbreak in Africa coincides with a massive Meningitis Vaccine campaign targeting “150 million Africans”, many throughout Guinea & Nigeria.

The cost-effective vaccine, MenAfriVac®, (less than US$0.50 per dose) was “kept outside the cold chain for up to four days at up to 40°C”.

Neighboring Liberia & Sierra Leone, both primary epicenters, were recently subject to the “largest ever Yellow Fever Immunization Program” conducted in that region – an estimated 12 million locals impacted (infected) by the compound shot.

Vaccine Resistance Movement is investigating the link between these extremely toxic experimental vaccines and the sudden surge in cases of Ebola.

Yellow Fever vaccine

Symptoms of ‘Acute fulminating Meningococcal Septicemia’, a virulent form of bacterial meningitis (marked by extreme vomiting, hemorrhaging – excessive bleeding around the eyes & mouth, severe blackish bruising on the arms & legs), leading to Septic shock and potentially even death, closely resemble those seen in Ebola victims.

This supposed outbreak of Ebola bares all the hallmarks of a rarified, virulent strain of bacterial Meningitis, ‘Acute fulminating Meningococcal Septicemia’, also known as ‘Waterhouse-Friderichsen Syndrome’.

Is this yet another case of a dangerous, untested vaccine triggering a tsunami of viral & bacterial mutations – in impoverished regions?

Waterhouse-Friderichsen Syndrome: ‘The prodromal symptoms were similar to those encountered in any respiratory infection, consisting of headache, chilly sensations, muscular pains and malaise. The onset of the bacteremia was sudden and dramatic. The most striking features were the profound shock and the petechial eruption, which in the course of a few hours became purpuric…This condition gradually progressed until numerous coarse, bubbling rales could be heard over both lung fields. With the appearance of frank pulmonary edema terminally, the patient lapsed into coma and died shortly thereafter.‘

Meningococcal Septicemia (Septic shock): ‘The hallmarks of severe sepsis and septic shock are changes that occur at the microvascular and cellular level with diffuse activation of inflammatory and coagulation cascades, vasodilation and vascular maldistribution, capillary endothelial leakage, and dysfunctional utilization of oxygen and nutrients at the cellular level. The challenge for clinicians is to recognize that this process is under way when it may not be clearly manifested in the vital signs or clinical examination.‘ Andre Kalil, MD, MPH Professor of Medicine, Department of Medicine, Section of Infectious Diseases; Director, Transplant ID Program, University of Nebraska Medical Center

Note: ‘DIC (Disseminated intravascular coagulation), is most commonly observed in severe sepsis and septic shock. Indeed, the development and severity of DIC correlate with mortality in severe sepsis. Although bacteremia, including both gram-positive and gram-negative organisms, is most commonly associated with DIC, other organisms (eg, viruses, fungi, and parasites) may also cause DIC.

DIC exists in both acute and chronic forms. Acute DIC develops when sudden exposure of blood to procoagulants (eg, tissue factor [TF], or tissue thromboplastin) generates intravascular coagulation. Compensatory hemostatic mechanisms are quickly overwhelmed, and, as a consequence, a severe consumptive coagulopathy leading to hemorrhage develops. Abnormalities of blood coagulation parameters are readily identified, and organ failure frequently results.‘ Marcel M Levi, MD Dean, Academic Medical Center, University of Amsterdam, The Netherlands

‘There is no evidence that MenAfriVac can cause meningococcal meningitis. Clinical alertness to the possibility of co-incidental meningitis should be maintained.‘ MenAfriVac® (Package insert)

Ebola virus

Ebola Virus: ‘Generally, the abrupt onset of Ebola haemorrhagic fever follows an incubation period of 2–21 days (mean 4–10) and is characterised by fever, chills, malaise, and myalgia. The subsequent signs and symptoms indicate multisystem involvement and include systemic (prostration), gastrointestinal (anorexia, nausea, vomiting, abdominal pain, diarrhoea), respiratory (chest pain, shortness of breath, cough, nasal discharge), vascular (conjunctival injection, postural hypotension, oedema), and neurological (headache, confusion, coma) manifestations.

Haemorrhagic manifestations arise during the peak of the illness and include petechiae, ecchymoses, uncontrolled oozing from venepuncture sites, mucosal haemorrhages, and post-mortem evidence of visceral haemorrhagic effusions. A macropapular rash associated with varying severity of erythema and desquamate can often be noted by day 5–7 of the illness; this symptom is a valuable differential diagnostic feature and is usually followed by desquamation in survivors. Abdominal pain is sometimes associated with hyperamylasaemia and true pancreatitis. In later stages, shock, convulsions, severe metabolic disturbances, and, in more than half the cases, diffuse coagulopathy supervene.‘

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(You read this kind of information and you are going to vote the criminals right back into office?)

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